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Scientists Discover HIV-Like “Mutation” Which Makes Coronavirus Extremely Infectious

February 26, 2020

Scientists Discover HIV-Like “Mutation” Which Makes Coronavirus Extremely Infectious

While mainstream scientists continue to perform mental gymnastics to insist that the new coronavirus wasn’t man-made, new research from scientists in China and Europe reveal that the disease happens to have an ‘HIV-like mutation’ which allows it to bind with human cells up to 1,000 times stronger than the Sars virus, according to SCMP.

Recall that at the end of January, a team of Indian scientists wrote in a now-retracted, scandalous paper claiming that the coronavirus may have been genetically engineered to incorporate parts of the HIV genome, writing “This uncanny similarity of novel inserts in the 2019- nCoV spike protein to HIV-1 gp120 and Gag is unlikely to be fortuitous in nature,” meaning – it was unlikely to have occurred naturally.

Fast forward to new research by a team from Nankai University, which writes that COV-19 has an ‘HIV-like mutation’ that  allows it to quickly enter the human body by binding with a receptor called ACE2 on a cell membrane.

Other highly contagious viruses, including HIV and Ebola, target an enzyme called furin, which works as a protein activator in the human body. Many proteins are inactive or dormant when they are produced and have to be “cut” at specific points to activate their various functions.

When looking at the genome sequence of the new coronavirus, Professor Ruan Jishou and his team at Nankai University in Tianjin found a section of mutated genes that did not exist in Sars, but were similar to those found in HIV and Ebola. –SCMP

“This finding suggests that 2019-nCoV [the new coronavirus] may be significantly different from the Sars coronavirus in the infection pathway,” reads the paper published this month on Chinaxiv.org – a platform used by the Chinese Academy of Sciences which releases research papers prior to peer-review.

…click on the above link to read the rest of the article…

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